Vertigo can be triggered by abnormalities in the central (brain stem, cerebellum, or brain) or peripheral (inner ear or vestibular nerve).
1. Physiologic
Vertigo physiological, is a condition caused by stimulation from the environment around the patient, which vestibule system, eyes, and cavernosometry still functioning well.
Included in this group are:
- Motion sickness. Motion sickness will be suppressed when out of sight around (visual surround) as opposed to the actual body movements. Motion sickness would be severe if the individual moves about the same direction with the movement of the body. Provoking circumstances are sitting in the back seat of a car, or reading book or note while car is moving.
- Space sickness. space sickness is a function of the state of weightlessness (weightlessness). In this situation there is a disruption of the balance between the semicircular canals and the otolith.
- Height vertigo is instability subjective, from postural and locomotor balance induced by visual, accompanied by the fear of falling, and vegetative symptoms.
2. Pathological Vertigo
Pathological Vertigo can be classified into:
a. Central, caused by abnormalities in the brain stem or cerebellum stem
b. Peripheral, caused by abnormalities in the inner ear or the vestibulocochlear cranial nerve (N. VIII)
c. Medical vertigo, can be caused by a decrease in blood pressure, hypoglicemia, or a metabolic disorder resulting from the treatment or systemic infection. Keywords for central vertigo resulting from a symptom or other brainstem signs or signs of acute onset deafness such as headaches and other neurological disorders such as trigeminal sensory loss the case, cebellar posteroinferior infarct artery. In these patients need to be quickly referred and investigated.
Red flag (alarm) in patients with vertigo include:
- Headache
- Neurologic symptoms
- Signs of neurological
It is also important to classify vertigo, be acute and chronic. Acute Vertigo usually have a single mechanism whereas chronic vertigo has a multifactorial mechanism. Chronic dizziness are more common in old age due to comorbid disease incidence is greater.
CENTRAL VERTIGO
Causes of central vertigo type, usually, there is a breakdown in the brain stem or cerebellum. To determine the disturbance in the brain stem, is there any other symptoms typical of the disorder in the brain stem, such as diplopia, paresthesias, changes in sensibility and motor function, weakness.
PERIPHERAL VERTIGO
The duration of vertigo:
a. Progresses in a few seconds. Most commonly caused by benign positional vertigo. Can be triggered by changes in head position. Progresses, a few seconds and then subsided. Most often the cause is idiopathic (unknown), but can also be caused by head trauma, ear surgery in vestibular neuronitis or by. Prognosis is generally good, the symptoms disappear spontaneously.
b. Progresses, a few minutes or hours. Can be found in Meniere's disease or recurrent vestibulopathy. Meniere's disease have a triad of symptoms that decreased auditory acuity (deaf), vertigo and tinnitus.
c. Progresses a few days to several weeks. Neuronitis vestibular, is a disorder that often come to the emergency unit. In this disease, onset of vertigo and nausea that accompanies it is also throwing up suddenly, and symptoms may progresses several days to several weeks. Auditory function was not impaired in vestibular neuronitis. On physical examination may be found nystagmus.
In addition we can distinguish between central and peripheral vertigo by nystagmus. Nystagmus is the eyeball movements that are involuntary, back and forth, rhythmic, with a certain frequency. Nystagmus is a form of reflex vestibulo oculer reaction against certain actions. Nystagmus can be physiological or pathological and manifest spontaneously or with tools such as test stimuli calories, tube spinning, spinning chairs, standing eyeball neutral or deviant or test positional or head movements
Vertigo may last only a few moments or may continue for several hours or even days. Patients often feel better when lying still, but vertigo can continue even if the patient does not move at all.
As per incident, vertigo there are several kinds:
a. Vertigo spontaneous. These Vertigo arise without giving stimuli. Stimuli arising from the illness itself, such as Meniere's disease because of the increasing Endolymph pressure. Vertigo a spontaneous component leads to the majors quickly glance both eyes.
b. Vertigo position. Vertigo is caused by changes in head position. Vertigo caused by stimulation of the semicircular canals cupula, by debris or cervical abnormalities. Debris is dirt on the cupula semicircular canals.
c. Vertigo calories. Vertigo is felt at the time of the calories. Vertigo is important to ask patients during caloric testing, so that he can compare this with the feeling of vertigo attacks ever experienced. When together, the complaint vertigonya is true, whereas if it is different, then the complaints of vertigo previously doubtful.
1. Physiologic
Vertigo physiological, is a condition caused by stimulation from the environment around the patient, which vestibule system, eyes, and cavernosometry still functioning well.
Included in this group are:
- Motion sickness. Motion sickness will be suppressed when out of sight around (visual surround) as opposed to the actual body movements. Motion sickness would be severe if the individual moves about the same direction with the movement of the body. Provoking circumstances are sitting in the back seat of a car, or reading book or note while car is moving.
- Space sickness. space sickness is a function of the state of weightlessness (weightlessness). In this situation there is a disruption of the balance between the semicircular canals and the otolith.
- Height vertigo is instability subjective, from postural and locomotor balance induced by visual, accompanied by the fear of falling, and vegetative symptoms.
2. Pathological Vertigo
Pathological Vertigo can be classified into:
a. Central, caused by abnormalities in the brain stem or cerebellum stem
b. Peripheral, caused by abnormalities in the inner ear or the vestibulocochlear cranial nerve (N. VIII)
c. Medical vertigo, can be caused by a decrease in blood pressure, hypoglicemia, or a metabolic disorder resulting from the treatment or systemic infection. Keywords for central vertigo resulting from a symptom or other brainstem signs or signs of acute onset deafness such as headaches and other neurological disorders such as trigeminal sensory loss the case, cebellar posteroinferior infarct artery. In these patients need to be quickly referred and investigated.
Red flag (alarm) in patients with vertigo include:
- Headache
- Neurologic symptoms
- Signs of neurological
It is also important to classify vertigo, be acute and chronic. Acute Vertigo usually have a single mechanism whereas chronic vertigo has a multifactorial mechanism. Chronic dizziness are more common in old age due to comorbid disease incidence is greater.
 |
| Characteristics of Vertigo |
CENTRAL VERTIGO
Causes of central vertigo type, usually, there is a breakdown in the brain stem or cerebellum. To determine the disturbance in the brain stem, is there any other symptoms typical of the disorder in the brain stem, such as diplopia, paresthesias, changes in sensibility and motor function, weakness.
PERIPHERAL VERTIGO
The duration of vertigo:
a. Progresses in a few seconds. Most commonly caused by benign positional vertigo. Can be triggered by changes in head position. Progresses, a few seconds and then subsided. Most often the cause is idiopathic (unknown), but can also be caused by head trauma, ear surgery in vestibular neuronitis or by. Prognosis is generally good, the symptoms disappear spontaneously.
b. Progresses, a few minutes or hours. Can be found in Meniere's disease or recurrent vestibulopathy. Meniere's disease have a triad of symptoms that decreased auditory acuity (deaf), vertigo and tinnitus.
c. Progresses a few days to several weeks. Neuronitis vestibular, is a disorder that often come to the emergency unit. In this disease, onset of vertigo and nausea that accompanies it is also throwing up suddenly, and symptoms may progresses several days to several weeks. Auditory function was not impaired in vestibular neuronitis. On physical examination may be found nystagmus.
In addition we can distinguish between central and peripheral vertigo by nystagmus. Nystagmus is the eyeball movements that are involuntary, back and forth, rhythmic, with a certain frequency. Nystagmus is a form of reflex vestibulo oculer reaction against certain actions. Nystagmus can be physiological or pathological and manifest spontaneously or with tools such as test stimuli calories, tube spinning, spinning chairs, standing eyeball neutral or deviant or test positional or head movements
Vertigo may last only a few moments or may continue for several hours or even days. Patients often feel better when lying still, but vertigo can continue even if the patient does not move at all.
As per incident, vertigo there are several kinds:
a. Vertigo spontaneous. These Vertigo arise without giving stimuli. Stimuli arising from the illness itself, such as Meniere's disease because of the increasing Endolymph pressure. Vertigo a spontaneous component leads to the majors quickly glance both eyes.
b. Vertigo position. Vertigo is caused by changes in head position. Vertigo caused by stimulation of the semicircular canals cupula, by debris or cervical abnormalities. Debris is dirt on the cupula semicircular canals.
c. Vertigo calories. Vertigo is felt at the time of the calories. Vertigo is important to ask patients during caloric testing, so that he can compare this with the feeling of vertigo attacks ever experienced. When together, the complaint vertigonya is true, whereas if it is different, then the complaints of vertigo previously doubtful.
No comments:
Post a Comment